Non-ciliary Roles of IFT Proteins in Cell Division and Polycystic Kidney Diseases

Benjamin Vitre; Audrey Guesdon; Benedicte Delaval

doi:10.3389/fcell.2020.578239

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Non-ciliary Roles of IFT Proteins in Cell Division and Polycystic Kidney Diseases

Journal article

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Non-ciliary Roles of IFT Proteins in Cell Division and Polycystic Kidney Diseases

Benjamin Vitre, Audrey Guesdon and Benedicte Delaval

Frontiers in Cell and Developmental Biology, Vol.8

22/09/2020

DOI: https://doi.org/10.3389/fcell.2020.578239

Abstract

Highlights d IFT proteins restrict proepicardial and myocardial development d IFT proteins act independently of primary cilia in this process d IFT proteins modulate BMP signaling by tuning Yap1-Tead activity d IFT88 and IFT20 interact with YAP1 in the cytoplasm to set Yap1-Tead activity

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Title: Non-ciliary Roles of IFT Proteins in Cell Division and Polycystic Kidney Diseases
Creators - without role: Benjamin Vitre - Université de Montpellier, Centre de Recherche en Biologie Cellulaire de Montpellier - CRBM
Audrey Guesdon - Université de Montpellier
Benedicte Delaval - Centre National de la Recherche Scientifique
Publication Details: Frontiers in Cell and Developmental Biology, Vol.8
Identifiers: 9947564109311
Academic Unit: Centre de Recherche en Biologie Cellulaire de Montpellier - CRBM
Language: English
Resource Type: Journal article
Local Fields: hal-02989220